Is Medication Causing Your Chronic Pain?

chronic pain

Many rely on medication to manage pain. Patients often come to me with the fear that the negative side-effects of these medications may be long-term. In this video, find out how this fear may be contributing to your chronic pain, and how to reverse this cycle.

 

One of the most important things to remember is that we are always in control of our thought patterns. Our reactions stick with us, while the effects of medication may be temporary.

Have you experienced unwanted side-effects, and worried that they’re now a part of your daily life? Break this cycle of pain, and join us on our journey to better health.

 

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Heather September 26, 2017

I enjoy your work however my studies in endocrinology, neurology, and developmental biology and biochemistry show that anti-epileptic medications actually can make long term changes to yourendocrin system as well the anti-epileptics have been shown to stop neuronal growth as well as stop you from building new neural networks which are needed to actually step out of pain. The gabba reuptake inhibitors are perhaps the worst offenders here. The actions do last for more than when you just take the pill because your body gets used to them being around if you take high doses three times a day. It’s Calle dupregulation. The cells make more receptors or less thereby either increasing or decreasing specificity. This means that if you took, for instance gabapentin 3 times a day for 5 years your neurons will have designed themselves to that dose due to the principales of equilibrium which is at the core of physiology. This is very important for patients to understand, that to adopt new ways of thinking, they will need to be able to build new neural networks which gabapentin class drugs do not allow you to do if you are at “therapeutic doses”. This is fairly new information from science which emerged from neuroscience and endocrinology over the last 5 years. It hasn’t been published in pain management circles yet but the information on the dangers of anti spasmodics for pain is now reaching the public sphere. I believe it is very important to address because they were inappropriate for use in non PTSD patients whose pain is not rooted in their brains and who need to leverage neuroplasticity in order to get out of the pain cycle. If a patient slowly titrates off these anti spasmodics, then after an adjustment period when the body can create new set points and metabolize and or create new receptors to readgjust for less gabba then they will be able to form new neural networks which are not infected with the pain message. I think the concept of neuroplasticity is where we can step out of the pain cycle and angrier spasmodics have been shown clinically and experimentally to stop this process. Gabba is the chief inhibitor neurotransmitter in the CNS, responsible for more than 1/3 of the brains function which is why the side effects are disastrous to people’s cortical function and cognitive ability. The great news is like you said, it will regenerate to normal after the patient goes off these drugs. Many of these anti-depressants are metabolized in the body and their chief effects occur in the gut where unwanted side effects occur. But when they act as hormones the effects are more lasting. The body is capable of amazing regenerative powers and it is sure up to the patient to develop a sense of control over their body and thoughts and mind through your wonderful program. I feel strongly however that this cannot happen while on certain drugs. I love your work and thank you so much for helping all of us! Much love an light

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